Disorders

Neutropenia, chronic granulomatous disease, hypereosinophilic syndrome, basophilia, and the granulocyte pathologies that matter clinically.

2026-09-10 · Hematology/Oncology Clinics of North America 2013
Chronic granulomatous disease.

Holland review of CGD: NADPH oxidase defects, infections, and management.

2026-09-10 · Blood 2015
How I treat hypereosinophilic syndromes.

Klion clinical approach to HES diagnosis, subtyping, and treatment.

2026-09-09 · New England Journal of Medicine 2003
A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome.

Cools et al. identified the FIP1L1-PDGFRA fusion in HES and showed it is the imatinib target.

2026-09-10 · Annals of the New York Academy of Sciences 2012
Leukocyte adhesion deficiencies.

Hanna and Etzioni review of LAD types, in which neutrophils cannot adhere and leave the blood.

2026-09-10 · Journal of Experimental Medicine 1999
Neutrophil-specific granule deficiency results from a novel mutation with loss of function of the transcription factor CCAAT/enhancer binding protein epsilon.

Lekstrom-Himes et al. traced specific granule deficiency to loss of C/EBP epsilon.

2026-09-09 · Molecular Oncology 2026
Hijacking emergency granulopoiesis: Neutrophil ontogeny and reprogramming in cancer.

Marinescu et al. review how tumors reshape emergency granulopoiesis and neutrophil programming.

2026-09-09 · Experimental Hematology 2026
Inherited disorders of granulopoiesis: understanding pathogenesis to advance new therapies.

Arreba-Tutusaus et al. review congenital neutropenias and their molecular basis.

2026-09-10 · Acta Haematol 2026
Complete Remission to Imatinib in a Myeloid Neoplasm with Basophilia and PRKG2::PDGFRA Gene Fusion: A Case Report.

Maddox et al. report a PRKG2::PDGFRA fusion myeloid neoplasm with basophilia that fully remitted on imatinib; single case.

2026-09-11 · Blood 2026
Neutrophil-dependent interferonopathy contributes to joint damage in hemophilia.

Recurrent synovial bleeding in severe hemophilia A drives a neutrophil-mediated type-I interferon response that accelerates hemophilic arthropathy.

2026-09-11 · Transplant Cell Ther 2026
Donor-derived distinct neutrophil subsets in G-CSF-mobilized peripheral blood grafts predict acute GVHD development.

Specific neutrophil subsets in G-CSF-mobilized allografts correlate with acute graft-versus-host disease risk after allo-HSCT.